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Moreover, a part of endothelial dysfunction has been proposed, which may contribute to abnormal vessel reactivity. However, testosterone in physiologic doses could even be beneficial in patients with angina , have larger ischemic thresholds and improved quality of life . Short-term intracoronary administration of testosterone induces coronary artery dilation and increases coronary blood circulate in males with established coronary artery disease . Therefore, an interplay of endogenous elements of the patient’s risk profile as nicely as the testosterone dosing could additionally be problems with whether or not adverse occasions happen.
We report a case of an acute ST-segment elevation myocardial infarction in a patient with conventional cardiac risk elements utilizing supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this affected person additionally had polycythemia, probably secondary to high-dose testosterone. The affected person underwent profitable percutaneous intervention of the proper coronary artery. Physicians and patients ought to concentrate on the medical penalties of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996).
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Rhoden EL, Morgentaler A. Risks of testosterone-replacement therapy and suggestions for monitoring. Testosterone alternative in hypogonadal men with angina improves ischaemic threshold and quality of life. Ajayi AAL, Mathur R, Halushka PV. Testosterone increase human platelet thromboxane A2 receptor density and aggregation responses.
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